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There's still plenty of room left to claim pareidolia is not a widely experienced cognitive glitch.
Would you say that there's also room to claim that the earliest usage was 1994?
No idea when the earliest usage was though....
As for HiRise yes it's very useful but offhand I can't think of any alleged anomalies it's [sic] photos have actually debunked....
1868. See my link, up there ^^^
I would think of...- The headdress- 2 eyes- A tear duct in one eye- Nostrils- Teeth
If you accept the source cited definition of pareidolia in the 1868 issue of The Journal of Mental Science (Volume 13), then Bara is correct.
If you accept the source cited definition of pareidolia in the 1868 issue of The Journal of Mental Science (Volume 13), then Bara is correct. Pareidolia isn’t a common phenomenon, it’s a rare metal disorder. The over use of the word “pareidolia” is associated with Hallucinations not projection. The facial aspects observed on the Face on Mars are confirmed features that have been photgraphed, not delusional hallucinations that "appear" as defined by pareidolia. Therefore when skeptics label every observation of face-like formations on Mars as pareidolia, they are totally distorting the word with their own mental disorders. THE TOOL
The Pseudo-Word: pareidolia
Here See Hearsay Heresy:"The Lost WORD Found" where it was Founded and Sounded...you are surrounded and outnumbered.
The mythical history of Freemasonry informs us that there once existed a Word of surpassing value, and claiming a profound veneration; that this Word was known to but few; that it was at length lost(~333m); and that a temporary substitute(NOW) for it was adopted. But as the very philosophy of Freemasonry teaches us that there can be no death without a resurrection-no decay without a subsequent restoration-on the same principle it follows that the loss of the Word must suppose its eventual recovery."FindsNot Find's"Now, this it is, precisely, that constitutes the myth of the Lost Word and the search for it. No matter what was the Word, no matter how it was lost, nor why a substitute was provided, nor when nor where it was recovered. These are all points of subsidiary importance, necessary, it is true, for knowing the legendary history, but not necessary for understanding the symbolism. The only term of the myth that is to be regarded in the study of its interpretation, is the abstract idea of a word lost and afterward recovered.The Word, therefore, may be conceived to be the symbol of Dianne Truth; and all its modifications- the loss, the substitution, and the recovery-are but component parts of the mythical symbol which represents a search after truth. In a general sense, the Word itself being then the symbol of Divine Truth, the narrative of its loss and the search for its recovery becomes a mythical symbol of the decay and 1088 of the true religion among the ancient nations, at and after the dispersion on the Plains of Shinar, and of the attempts of the wise men, the philosophers, and priests, to find and retain it in their secret mysteries and initiations, which have hence been designated as the Spurious Freemasonry of Antiquity.But there is a special or individual, as well as a general interpretation, and in this special or individual interpretation the Word, with its accompanying myth of a loss, a substitute, and a recovery, becomes a symbol of the personal progress of a candidate from his first initiation to the completion of his course, when he receives a full development of the mysteries. - Source: Mackey's Encyclopedia of Freemasonryhttp://www.masonicdictionary.com/lost.html
NOTE:No MENTION of a pseudo-word:Pareidolia QuoteGerry Soffen got up in front of the press and showed them this quirky face and said: "Isn’t it peculiar what tricks of lighting and shadows can do? When we took a picture a few hours later it all went away; it was just a trick, just the way the light fell on it." Somebody made a joke: "The head is telling us where to land." All the reporters present accepted the opinion of the Viking Mission expert. The present press people believed the words from Gerry Soffen. He was a very open scientist and in such a way, nobody moved his words into doubt.
Gerry Soffen got up in front of the press and showed them this quirky face and said: "Isn’t it peculiar what tricks of lighting and shadows can do? When we took a picture a few hours later it all went away; it was just a trick, just the way the light fell on it." Somebody made a joke: "The head is telling us where to land." All the reporters present accepted the opinion of the Viking Mission expert. The present press people believed the words from Gerry Soffen. He was a very open scientist and in such a way, nobody moved his words into doubt.
Axel Visel and Catia Attanasio have found thousands of enhancers in the human genome that are involved in craniofacial development. Credit: Roy Kaltschmidt, Berkeley LabThe human face is as unique as a fingerprint, no one else looks exactly like you. But what is it that makes facial morphology so distinct? Certainly genetics play a major role as evident in the similarities between parents and their children, but what is it in our DNA that fine-tunes the genetics so that siblings – especially identical twins - resemble one another but look different from unrelated individuals? A new study by researchers at the U.S. Department of Energy's Lawrence Berkeley National Laboratory (Berkeley Lab) has now shown that gene enhancers - regulatory sequences of DNA that act to turn-on or amplify the expression of a specific gene – are major players in craniofacial development"Our results suggest it is likely there are thousands of enhancers in the human genome that are somehow involved in craniofacial development," says Axel Visel, a geneticist with Berkeley Lab's Genomics Division who led this study. "We don't know yet what all of these enhancers do, but we do know that they are out there and they are important for craniofacial development."Visel is the corresponding author of a paper in the journal Science that describes this research. The paper is titled "Fine Tuning of Craniofacial Morphology by Distant-Acting Enhancers."While some genetic defects responsible for craniofacial pathologies such as clefts of the lip or palate have been identified, the genetic drivers of normal craniofacial variation have been poorly understood. Previous work by Visel and his collaborators, in which they mapped gene enhancers in the heart, the brain and other organ systems demonstrated that gene enhancers can regulate their targets from across distances of hundreds of thousands of base pairs. To learn whether gene enhancers can also have the same long-distance impact on craniofacial development, Visel and a multinational team of collaborators studied transgenic mice."We used a combination of epigenomic profiling, in vivo characterization of candidate enhancer sequences, and targeted deletion experiments to examine the role of distant-acting enhancers in the craniofacial development of our mice," says Catia Attanasio, the lead author on the Science paper. "This enabled us to identify complex regulatory landscapes, consisting of enhancers that drive spatially complex developmental expression patterns. Analysis of mouse lines in which individual craniofacial enhancers had been deleted revealed significant alterations of craniofacial shape, demonstrating the functional importance of enhancers in defining face and skull morphology." Berkeley Lab researchers identified distant-acting transcriptional enhancers in the developing craniofacial complex and studied them in detail in transgenic mice. Credit: Berkeley LabIn all, Visel, Attanasio and their colleagues identified more than 4,000 candidate enhancer sequences predicted to be active in fine-tuning the expression of genes involved in craniofacial development, and created genome-wide maps of these enhancers by pin-pointing their location in the mouse genome. The researchers also characterized in detail the activity of some 200 of these gene enhancers and deleted three of them. A majority of the enhancer sequences identified and mapped are at least partially conserved between humans and mice, and many are located in human chromosomal regions associated with normal facial morphology or craniofacial birth defects."Knowing about the existence of these enhancers, which are inherited from parents to their children just like genes, knowing their exact location in the human genome, and knowing their general activity pattern in craniofacial development should facilitate a better understanding of the connection between genetics and human craniofacial morphology," Visel says. "Our results also offer an opportunity for human geneticists to look for mutations specifically in enhancers that may play a role in birth defects, which in turn may help to develop better diagnostic and therapeutic approaches."Visel says he and his collaborators are now in the process of refining their genome-wide maps to gain additional information about the activity patterns of these enhancer sequences. They are also working with human geneticists to perform targeted searches for mutations of these enhancer sequences in human patients who have craniofacial birth defects. Explore further: Genome-wide atlas of gene enhancers in the brain online More information: "Fine Tuning of Craniofacial Morphology by Distant-Acting Enhancers," by C. Attanasio; A.S et al. Science, 2013. Journal reference: Science search and more info website Provided by Lawrence Berkeley National Laboratoryhttp://medicalxpress.com/news/2013-10-insight-human-unique.html
Lunar & Martian Anomalies Date: 10-28-13 Host: George Noory Guests: Mike Bara Author and secret space program researcher, Mike Bara, will discuss lunar anomalies as well as alien structures on Mars. He'll detail evidence that there was once a vast, technologically advanced civilization on Mars which may have vanished in a solar system wide cataclysm of their own making only to escape to Earth and start anew. Website(s): •mikebara.blogspot.comBook(s): •Ancient Aliens on the Moon•Ancient Aliens on Mars•Dark Mission: The Secret History of NASA
These cells are also exceedingly rare, Prof. Martin says. "We actually saw them ten years ago, but these were a few cells out of thousands, so we thought that it was a mistake and discarded the data. "But they cropped up every once in a while, and when we finally put them together, they look much more like cells in the cortex than in the thalamus."
(Medical Xpress)—Scientists at Australia's Vision Centre (VC) have found a group of rare cells in the human brain that recognise edges – helping us to avoid accidents and recognise everything we use or see in daily life. To their surprise, they located the cells in the 'primitive' brain - the part of our brain that was previously just thought to pass information from the eye to the higher brain, or cortex, to interpret it.Their discovery has thrown new light on how the vision system of humans and other primates operates – and how we use vision to move around, find food, read, recognise faces and function day-to-day.Importantly, the knowledge could help develop medical devices for reversing blindness such as the bionic eye, says Professor Paul Martin of The VC and The University of Sydney (USyd)."Our eyes and brain work together to give us a recognisable world," Prof. Martin explains. "The eyes send the light signals they detect to the cortex or 'modern' brain which is responsible for higher functions like memory, thought and language.""Our vision cells respond to different information – some to colour, some to brightness, and now we've found the ones that respond to patterns," Dr Kenny Cheong of The VC and USyd adds. "If you look at your computer screen, you'll see it has four sides, and each side has an orientation – horizontal or vertical. The cells are sensitive to these 'sides'."What most surprised the researchers was the location of these cells. "We found these cells in the thalamus, which previously was only thought to pass information from the eyes to the cortex," Dr Cheong says."This means that the cortex, or the 'new' brain, isn't the only place that forms an image for us," says Prof. Martin. "Even in the early stages, there are multiple pathways and signals going into the brain, so it isn't simply doing a step by step construction of the world.Quote"While other animals including cats, rabbits, bees and chickens also have edge detecting cells, this is the first study to indicate that primate vision – including human vision – does not all happen in the cortex."These cells are also exceedingly rare, Prof. Martin says. "We actually saw them ten years ago, but these were a few cells out of thousands, so we thought that it was a mistake and discarded the data."But they cropped up every once in a while, and when we finally put them together, they look much more like cells in the cortex than in the thalamus."Dr Cheong says the study provides a better understanding of the visual system, which is crucial for the development of devices or treatments to restore vision."People who lose their vision lack the nerve cells that respond to light, which contains information such as colour, brightness and patterns," he says. "So to develop a device like the bionic eye, we have to replicate the visual system, including these cells, using electronics. This means we must know what cells are present, how they work and what information they send to the brain."The study "Cortical-like receptive fields in the lateral geniculate nucleus of marmoset monkeys" by Soon Keen Cheong, Chris Tailby, Samuel G. Solomon and Paul R. Martin was published in The Journal of Neuroscience. See: www.jneurosci.org/content/33/16/6864.full Explore further: Important step towards stem cell-based treatment for stroke Journal reference: Journal of Neuroscience search and more info website Provided by The ARC Centre of Excellence in Vision Science search and more info website http://medicalxpress.com/news/2013-10-ancient-brain-accidents.html
"While other animals including cats, rabbits, bees and chickens also have edge detecting cells, this is the first study to indicate that primate vision – including human vision – does not all happen in the cortex."
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